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  1. Carolyn Widney Greider (born April 15, 1961) is an American molecular biologist and Nobel laureate. She joined the University of California, Santa Cruz as a Distinguished Professor in the department of molecular, cell, and developmental biology [1] in October 2020.

    • Molecular biology
    • Discovery of telomerase
  2. Carol Widney Greider (* 15. April 1961 in San Diego, Kalifornien, USA) ist eine US-amerikanische Molekularbiologin, die durch ihre Arbeiten über das Enzym Telomerase bekannt wurde. Ihr wurde zusammen mit Elizabeth H. Blackburn und Jack W. Szostak der Nobelpreis für Physiologie oder Medizin für 2009 zugesprochen. [1] Inhaltsverzeichnis.

  3. Carol W. Greider. The Nobel Prize in Physiology or Medicine 2009. Born: 15 April 1961, San Diego, CA, USA. Affiliation at the time of the award: Johns Hopkins University School of Medicine, Baltimore, MD, USA. Prize motivation: “for the discovery of how chromosomes are protected by telomeres and the enzyme telomerase” Prize share: 1/3. Life.

  4. 2. Mai 2024 · Carol W. Greider, American molecular biologist who was awarded the 2009 Nobel Prize for Physiology or Medicine, along with Elizabeth H. Blackburn and Jack W. Szostak, for her research into telomeres and for her discovery of an enzyme called telomerase. Learn more about Greiders life and work.

    • Kara Rogers
  5. Carol Greider is a professor of molecular biology and genetics at Johns Hopkins University and a 2009 Nobel Prize winner for her discovery of telomerase, an enzyme that maintains chromosome ends. Learn about her research, her journey and her insights on basic and curiosity-driven science.

  6. The Nobel Prize in Physiology or Medicine 2009 was awarded jointly to Elizabeth H. Blackburn, Carol W. Greider and Jack W. Szostak "for the discovery of how chromosomes are protected by telomeres and the enzyme telomerase"

  7. University Professor of Molecular Biology and Genetics. cgreider@jhmi.edu. 603 PCTB, 725 N. Wolfe Street. Baltimore, MD 21205-2185. Publications. Lab website. Telomeres protect chromosome ends from being recognized as DNA damage and chromosomal rearrangements.